<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>0212-1611</journal-id>
<journal-title><![CDATA[Nutrición Hospitalaria]]></journal-title>
<abbrev-journal-title><![CDATA[Nutr. Hosp.]]></abbrev-journal-title>
<issn>0212-1611</issn>
<publisher>
<publisher-name><![CDATA[Grupo Arán]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S0212-16112018001200011</article-id>
<article-id pub-id-type="doi">10.20960/nh.2287</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[El yogur y recomendaciones dietéticas en la intolerancia a la lactosa]]></article-title>
<article-title xml:lang="en"><![CDATA[Yogurt and dietary recommendations for lactose intolerance]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Saborido]]></surname>
<given-names><![CDATA[Rebeca]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Leis]]></surname>
<given-names><![CDATA[Rosaura]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Hospital Clínico Universitario de Santiago de Compostela-Xerencia de Xestión Integrada de Santiago. USC Departamento de Pediatría Unidad de Gastroenterologia, Hepatología y Nutrición Infantil]]></institution>
<addr-line><![CDATA[Santiago de Compostela A Coruña]]></addr-line>
<country>Spain</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>00</month>
<year>2018</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>00</month>
<year>2018</year>
</pub-date>
<volume>35</volume>
<numero>spe6</numero>
<fpage>45</fpage>
<lpage>48</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://scielo.isciii.es/scielo.php?script=sci_arttext&amp;pid=S0212-16112018001200011&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://scielo.isciii.es/scielo.php?script=sci_abstract&amp;pid=S0212-16112018001200011&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://scielo.isciii.es/scielo.php?script=sci_pdf&amp;pid=S0212-16112018001200011&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen La malabsorción de la lactosa se produce por la incapacidad para la digestión del azúcar debido a la disminución de la actividad de la lactasa intestinal tras un desorden primario o secundario a otras patologías. La hipolactasia primaria tipo adulto es un trastorno autosómico recesivo, caracterizado por la pérdida progresiva de lactasa tras el destete, mientras que la secundaria es un trastorno transitorio que se corregirá tras la curación de la patología de base. Para el diagnóstico de la malabsorción a la lactosa destacan los test de hidrógeno y metano espirado tras sobrecarga. En el déficit primario tardío de lactasa puede realizarse el test molecular del polimorfismo de nucleótido simple (SNP C/T-13910). El diagnóstico de la intolerancia precisa de la presencia de sintomatología tras el consumo de lactosa. El tratamiento de la hipolactasia primaria tipo adulto consiste en disminuir la lactosa de la dieta por debajo de la dosis gatillo. Un porcentaje importante de individuos con malabsorción toleran cantidades habituales de consumo y prácticamente el 99% toleran yogur o derivados lácteos fermentados, lo que permite así cubrir las recomendaciones diarias de ingesta de calcio y vitamina D. Además, estrategias nutricionales que reduzcan la carga de lactosa, el tiempo de vaciamiento gástrico y/o el tiempo de tránsito intestinal o que incrementen la actividad lactásica y la compensación colónica van a permitir una mayor tolerancia.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract Malabsorption to lactose is caused by the inability to digest sugar due to the decrease in the activity of intestinal lactase. Malabsorption may be due to a primary or secondary disorder. Adult type primary hypolactasia is an autosomal recessive disorder, characterized by the progressive loss of lactase after weaning. The secondary hypolactasia is a transitory disorder, which will be corrected after the cure of the basic pathology. For lactose malabsorption diagnosis, the hydrogen and methane exhaled tests after lactose overload stand out and, in the case of the primary adult type, the molecular test of the simple nucleotide polymorphism (SNP C / T-13910). However, the diagnosis of lactose intolerance requires the presence of symptoms after consumption. The treatment of primary adult-type hypolactasia consists in decreasing the lactose in the diet below the trigger dose. A significant percentage of individuals with malabsorption tolerate habitual amounts of consumption. Practically 99% of them tolerate yogurt or fermented dairy products, thus allowing to cover the daily recommendations of calcium and vitamin D intake. In addition, nutritional strategies that reduce the lactose load, gastric emptying time and / or intestinal transit time or increase lactic activity and colonic compensation, will allow a greater tolerance.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[Lactosa]]></kwd>
<kwd lng="es"><![CDATA[Lactasa]]></kwd>
<kwd lng="es"><![CDATA[Malabsorción]]></kwd>
<kwd lng="es"><![CDATA[Intolerancia]]></kwd>
<kwd lng="es"><![CDATA[Yogur]]></kwd>
<kwd lng="en"><![CDATA[Lactose]]></kwd>
<kwd lng="en"><![CDATA[Lactase]]></kwd>
<kwd lng="en"><![CDATA[Malabsorption]]></kwd>
<kwd lng="en"><![CDATA[Intolerance]]></kwd>
<kwd lng="en"><![CDATA[Yogurt]]></kwd>
</kwd-group>
</article-meta>
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