<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>0212-1611</journal-id>
<journal-title><![CDATA[Nutrición Hospitalaria]]></journal-title>
<abbrev-journal-title><![CDATA[Nutr. Hosp.]]></abbrev-journal-title>
<issn>0212-1611</issn>
<publisher>
<publisher-name><![CDATA[Grupo Arán]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S0212-16112019000600025</article-id>
<article-id pub-id-type="doi">10.20960/nh.02673</article-id>
<title-group>
<article-title xml:lang="en"><![CDATA[Diet, physical activity and telomere length in adults]]></article-title>
<article-title xml:lang="es"><![CDATA[Dieta, actividad física y longitud telomérica en adultos]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Navarro Ibarra]]></surname>
<given-names><![CDATA[María Jossé]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Hernández]]></surname>
<given-names><![CDATA[Jesús]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Caire Juvera]]></surname>
<given-names><![CDATA[Graciela]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Centro de Investigación en Alimentación y Desarrollo A.C. Department of Nutrition ]]></institution>
<addr-line><![CDATA[Hermosillo Sonora]]></addr-line>
<country>Mexico</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>12</month>
<year>2019</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>12</month>
<year>2019</year>
</pub-date>
<volume>36</volume>
<numero>6</numero>
<fpage>1403</fpage>
<lpage>1417</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://scielo.isciii.es/scielo.php?script=sci_arttext&amp;pid=S0212-16112019000600025&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://scielo.isciii.es/scielo.php?script=sci_abstract&amp;pid=S0212-16112019000600025&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://scielo.isciii.es/scielo.php?script=sci_pdf&amp;pid=S0212-16112019000600025&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract Telomere length (TL) is a predictive biomarker of premature aging. Telomere shortening has been linked to age-related diseases and noncommunicable diseases (NCD), and may reflect the effects of behavioral, psychosocial and environmental factors on health status. Telomere attrition can be affected by lifestyle factors such as diet and physical activity. The search of studies included in this review was conducted on PubMed Central database. A majority of studies are cross-sectional, as there is a clear lack of prospective studies to evaluate the individual effect of dietary components, dietary patterns, and physical activity on TL in the long term. The current literature suggests that high adherence to Mediterranean diet (MD), with consumption of antioxidants, fiber and vegetables, as well as seeds and walnuts, is associated with longer TL. The dietary components of a healthy diet, such as carotenoids, vitamins A, C, D, E, polyphenols, fiber, and omega-3 fatty acids could help maintain TL. In contrast, a high consumption of sugary beverages, processed meat, and proinflammatory diets is associated with telomere shortening. In a majority of studies TL is positively associated with moderate physical activity. The predominant mechanisms through which a healthy diet and moderate physical exercise could mitigate telomere attrition include decreasing oxidative stress and inflammation. We shall not discuss the associations of possible risk or protective factors in terms of causality since the majority of studies are cross-sectional and randomized controlled trials are limited; accordingly, some results are inconclusive. For future research, we suggest evaluating the individual effects of dietary components, dietary patterns and physical activity, considering repeated measurements and exercise intensity, on TL. It is also advisable to include biomarkers of oxidative stress and inflammation proteins, and to measure telomerase activity.]]></p></abstract>
<abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen La longitud de los telómeros (TL) es un biomarcador predictivo del envejecimiento prematuro. El acortamiento de los telómeros se ha relacionado con las enfermedades asociadas a la edad y las enfermedades no transmisibles (ENT), y puede reflejar los efectos de los factores conductuales, psicosociales y ambientales en el estado de salud. El desgaste de los telómeros puede verse afectado por factores del estilo de vida, como la dieta y la actividad física. La búsqueda de los estudios incluidos en esta revisión se realizó en la base de datos PubMed Central. La mayoría de los estudios son transversales, por lo que está clara la falta de estudios prospectivos que evalúen el efecto individual de los componentes dietéticos, los patrones dietéticos y la actividad física sobre el TL a largo plazo. La literatura actual sugiere que una alta adherencia a la dieta mediterránea (DM) y el consumo de antioxidantes, fibra y vegetales, así como semillas y nueces, se asocia a una mayor TL. Los componentes dietéticos de una dieta saludable, como los carotenoides, las vitaminas A, C, D, E, los polifenoles, la fibra y los ácidos grasos omega-3, podrían ayudar a mantener la TL. En contraste, el alto consumo de bebidas azucaradas, carne procesada y dietas proinflamatorias se asocia al acortamiento de los telómeros. En la mayoría de los estudios, el TL se asocia positivamente con la actividad física moderada. Los mecanismos predominantes que, a través de una dieta saludable y un ejercicio físico moderado, podrían mitigar el desgaste de los telómeros son la disminución del estrés oxidativo y la inflamación. No se discute la asociación de posibles factores de riesgo o de protección en términos de causalidad, ya que la mayoría de los estudios son transversales y los ensayos controlados aleatorios son limitados; por consiguiente, algunos resultados no son concluyentes. Para investigaciones futuras se sugiere evaluar los efectos individuales de los componentes dietéticos, los patrones de actividad física y dietética, considerando mediciones repetidas y la intensidad del ejercicio, sobre el TL. También es aconsejable incluir biomarcadores de estrés oxidativo y proteínas inflamatorias, y medir la actividad de la telomerasa.]]></p></abstract>
<kwd-group>
<kwd lng="en"><![CDATA[Telomere length]]></kwd>
<kwd lng="en"><![CDATA[Diet]]></kwd>
<kwd lng="en"><![CDATA[Physical activity]]></kwd>
<kwd lng="es"><![CDATA[Longitud de los telómeros]]></kwd>
<kwd lng="es"><![CDATA[Dieta]]></kwd>
<kwd lng="es"><![CDATA[Actividad física]]></kwd>
</kwd-group>
</article-meta>
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