<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>0212-1611</journal-id>
<journal-title><![CDATA[Nutrición Hospitalaria]]></journal-title>
<abbrev-journal-title><![CDATA[Nutr. Hosp.]]></abbrev-journal-title>
<issn>0212-1611</issn>
<publisher>
<publisher-name><![CDATA[Grupo Arán]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S0212-16112025000200013</article-id>
<article-id pub-id-type="doi">10.20960/nh.05541</article-id>
<title-group>
<article-title xml:lang="en"><![CDATA[Causal effects of vitamin D on leukemia risk: insights from two-sample Mendelian randomization analysis]]></article-title>
<article-title xml:lang="es"><![CDATA[Efectos causales de la vitamina D sobre el riesgo de leucemia: aportaciones del análisis de aleatorización mendeliana de dos muestras]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Chen]]></surname>
<given-names><![CDATA[Shupeng]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Zhang]]></surname>
<given-names><![CDATA[Meiling]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Gao]]></surname>
<given-names><![CDATA[Yao]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Zeng]]></surname>
<given-names><![CDATA[Yingjian]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,School of Clinical Medicine  ]]></institution>
<addr-line><![CDATA[Nanchang ]]></addr-line>
<country>People's Republic of China</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Jiangxi University of Traditional Chinese Medicine Affiliated Hospital ]]></institution>
<addr-line><![CDATA[Nanchang ]]></addr-line>
<country>People's Republic of China</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>04</month>
<year>2025</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>04</month>
<year>2025</year>
</pub-date>
<volume>42</volume>
<numero>2</numero>
<fpage>333</fpage>
<lpage>340</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://scielo.isciii.es/scielo.php?script=sci_arttext&amp;pid=S0212-16112025000200013&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://scielo.isciii.es/scielo.php?script=sci_abstract&amp;pid=S0212-16112025000200013&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://scielo.isciii.es/scielo.php?script=sci_pdf&amp;pid=S0212-16112025000200013&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract  Background: vitamin D plays a crucial role in immune regulation, anti-inflammatory processes, and tumor suppression, but its relationship with leukemia risk remains unclear. This study aims to evaluate the causal relationship between vitamin D levels and the risk of different types of leukemia through a two-sample Mendelian randomization (MR) analysis.  Methods: data from large-scale genome-wide association studies (GWAS) were used, and genetic variants associated with vitamin D were selected as instrumental variables. The relationship between vitamin D levels and the risk of acute myeloid leukemia (AML), acute lymphoblastic leukemia (ALL), chronic lymphocytic leukemia (CLL), and chronic myeloid leukemia (CML) was examined. The inverse variance weighted (IVW) method was applied as the primary analytical approach. Heterogeneity was assessed through Cochran's Q test, pleiotropy was evaluated using the MR-Egger intercept, and sensitivity analyses were performed to ensure the robustness of the results.  Results: MR analysis showed a significant inverse association between serum 25-hydroxyvitamin D levels and the risk of CML (OR = 0.44, 95 % CI: 0.25-0.78, p = 0.005), suggesting a potential protective effect of vitamin D against CML. No significant causal relationships were found between vitamin D levels and the risks of AML, ALL, or CLL. Sensitivity analyses supported the robustness of these findings, with no evidence of heterogeneity or pleiotropy.  Conclusion: the findings indicate that higher vitamin D levels may reduce the risk of CML, while the effects on other types of leukemia require further investigation. The potential role of vitamin D in leukemia prevention warrants more mechanistic studies and clinical validation.]]></p></abstract>
<abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen  Antecedentes: la vitamina D juega un papel vital en la inmunomodulación, los procesos antiinflamatorios y la inhibición tumoral, pero su relación con el riesgo de leucemia no está clara. El objetivo de este estudio es evaluar la relación causal entre los niveles de vitamina D y el riesgo de diferentes tipos de leucemia mediante un análisis aleatorizado de Mendel (MR) de dos muestras.  Método: utilizando datos de estudios de asociaciones pangenómicas a gran escala (GWAS) se seleccionaron como variables instrumentales las variaciones genéticas relacionadas con la vitamina D. Se estudió la relación entre los niveles de vitamina D y el riesgo de leucemia mieloide aguda (LMA), leucemia linfoblástica aguda (LLA), leucemia linfoblástica crónica (LLC) y leucemia mieloide crónica. El método de ponderación de variación inversa (IVW) se utiliza como principal método de análisis. La heterogeneidad se evalúa a través de la prueba de la Q de Cochran; la polivalencia se evalúa con la interceptación MR-Egger y se realiza un análisis de sensibilidad para garantizar la solidez de los resultados.  Resultados: el análisis de MR mostró una correlación negativa significativa entre los niveles séricos de 25-hidroxivitamina D y el riesgo de CML (OR = 0,44, IC 95 %: 0,25-0,78, p = 0,005), lo que indica que la vitamina D tiene un potencial efecto protector sobre la LMC. No se encontró una relación causal significativa entre los niveles de vitamina D y el riesgo de LMA, LLA o LLC. El análisis de sensibilidad apoya la solidez de estos hallazgos y no hay evidencia de heterogeneidad o polivalencia.  Conclusiones: los resultados del estudio sugieren que los niveles más altos de vitamina D pueden reducir el riesgo de LMC mientras que los efectos en otros tipos de leucemia requieren más estudios. El papel potencial de la vitamina D en la prevención de la leucemia merece más investigación de mecanismos y verificación clínica.]]></p></abstract>
<kwd-group>
<kwd lng="en"><![CDATA[Vitamin D]]></kwd>
<kwd lng="en"><![CDATA[Leukemia]]></kwd>
<kwd lng="en"><![CDATA[Mendelian randomization]]></kwd>
<kwd lng="en"><![CDATA[CML]]></kwd>
<kwd lng="en"><![CDATA[Causal relationship]]></kwd>
<kwd lng="en"><![CDATA[Genome-wide association studies (GWAS)]]></kwd>
<kwd lng="es"><![CDATA[Vitamina D]]></kwd>
<kwd lng="es"><![CDATA[Leucemia]]></kwd>
<kwd lng="es"><![CDATA[Aleatorización mendeliana]]></kwd>
<kwd lng="es"><![CDATA[Leucemia mieloide crónica]]></kwd>
<kwd lng="es"><![CDATA[Relación causal]]></kwd>
<kwd lng="es"><![CDATA[Estudio de asociación pangenómica (GWAS)]]></kwd>
</kwd-group>
</article-meta>
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