<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>1889-836X</journal-id>
<journal-title><![CDATA[Revista de Osteoporosis y Metabolismo Mineral]]></journal-title>
<abbrev-journal-title><![CDATA[Rev Osteoporos Metab Miner]]></abbrev-journal-title>
<issn>1889-836X</issn>
<publisher>
<publisher-name><![CDATA[Sociedad Española de Investigaciones Óseas y Metabolismo Mineral]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S1889-836X2026000100003</article-id>
<article-id pub-id-type="doi">10.20960/revosteoporosmetabminer.00090</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Relación del hábito tabáquico con la prevalencia y progresión de la calcificación aórtica abdominal en hombres]]></article-title>
<article-title xml:lang="en"><![CDATA[Relationship between smoking status and the prevalence and progression of abdominal aortic calcification in men]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Villabrille]]></surname>
<given-names><![CDATA[Sara Fernández]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
<xref ref-type="aff" rid="Aaf"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Alonso]]></surname>
<given-names><![CDATA[Carlos Gómez]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
<xref ref-type="aff" rid="Aaf"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Antequera]]></surname>
<given-names><![CDATA[Carmen Palomo]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
<xref ref-type="aff" rid="Aaf"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[García]]></surname>
<given-names><![CDATA[Minerva Rodríguez]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
<xref ref-type="aff" rid="Aaf"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[López]]></surname>
<given-names><![CDATA[Teresa Naves]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Faedo]]></surname>
<given-names><![CDATA[Mónica Llaneza]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Mendívil]]></surname>
<given-names><![CDATA[Laura Naves]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Muñoz]]></surname>
<given-names><![CDATA[Javier Montero]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
<xref ref-type="aff" rid="Aaf"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Díaz]]></surname>
<given-names><![CDATA[Manuel Naves]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
<xref ref-type="aff" rid="Aaf"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Instituto de Investigación Sanitaria del Principado de Asturias (ISPA) Hospital Universitario Central de Asturias. Universidad de Oviedo Unidad de Metabolismo Óseo. Unidad de Gestión Clínica de Medicina Interna]]></institution>
<addr-line><![CDATA[Oviedo ]]></addr-line>
<country>Spain</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Instituto de Investigación Sanitaria del Principado de Asturias (ISPA) Hospital Universitario Central de Asturias. Universidad de Oviedo Unidad de Gestión Clínica de Medicina Interna]]></institution>
<addr-line><![CDATA[Oviedo ]]></addr-line>
<country>Spain</country>
</aff>
<aff id="Af3">
<institution><![CDATA[,Instituto de Investigación Sanitaria del Principado de Asturias (ISPA) Hospital Universitario Central de Asturias. Universidad de Oviedo Área Gestión Clínica de Nefrología]]></institution>
<addr-line><![CDATA[Oviedo ]]></addr-line>
<country>Spain</country>
</aff>
<aff id="Af4">
<institution><![CDATA[,Instituto de Investigación Sanitaria del Principado de Asturias (ISPA) Hospital Monte Naranco. Universidad de Oviedo Servicio de Geriatría]]></institution>
<addr-line><![CDATA[Oviedo ]]></addr-line>
<country>Spain</country>
</aff>
<aff id="Af5">
<institution><![CDATA[,RICORS2040-RENAL (RD24/0004/006) RICORS2040-RENAL (RD24/0004/006) ]]></institution>
<addr-line><![CDATA[Oviedo ]]></addr-line>
<country>Spain</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>03</month>
<year>2026</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>03</month>
<year>2026</year>
</pub-date>
<volume>18</volume>
<numero>1</numero>
<fpage>3</fpage>
<lpage>11</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://scielo.isciii.es/scielo.php?script=sci_arttext&amp;pid=S1889-836X2026000100003&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://scielo.isciii.es/scielo.php?script=sci_abstract&amp;pid=S1889-836X2026000100003&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://scielo.isciii.es/scielo.php?script=sci_pdf&amp;pid=S1889-836X2026000100003&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen  Introducción y objetivo: la calcificación aórtica abdominal (CAA) suele preceder al desarrollo de calcificación de la arteria coronaria, contribuye a rigidez arterial y predice eventos cardiovasculares y mortalidad. El objetivo del trabajo fue valorar la asociación entre el hábito tabáquico y la presencia y progresión de CAA en población masculina.  Material y métodos: se seleccionaron 306 hombres &gt; 50 años que realizaron un estudio radiológico dorsolumbar, repetido 4 años después, para determinar la presencia y progresión de CAA. Se recogieron entre otros, edad, índice de masa corporal (IMC), diabetes y hábito tabáquico: edad de inicio, cigarrillos/día fumados y años de duración.  Resultados: los fumadores y exfumadores tuvieron mayor prevalencia de CAA (52,7 % y 52,6 %) que los no fumadores (25 %), siendo de mayor edad los exfumadores. Ajustado por edad e IMC, ser fumador se asoció con prevalencia de CAA (odds ratio [OR] = 3,95; intervalo de confianza del 95 % [IC 95 %], 2,00-7,82). Similar asociación se encontró en exfumadores (OR = 2,93; IC 95 %: 1,56-5,51). Diez años de envejecimiento se asociaron con CAA (OR = 1,64; IC 95 %: 1,35-1,93), riesgo 4,6 y 3 veces inferior al de ser fumador o exfumador. Tras 4 años de seguimiento, ser fumador y exfumador se asoció con progresión de CAA (OR = 2,57; IC 95 %, 1,08-6,14) y (OR = 2,34; IC 95 %, 1,07-5,13) respectivamente. El alto consumo tabáquico y tiempo de consumo modificaron estos resultados hacia un riesgo mayor.  Conclusiones: el hábito tabáquico se relacionó con una mayor prevalencia y progresión de CAA. En conclusión, el hábito tabáquico se asoció con mayor riesgo de CAA que el propio envejecimiento.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract  Introduction and objective: abdominal aortic calcification (AAC) often precedes the development of coronary artery calcification, contributes to arterial stiffness, and predicts cardiovascular events and mortality. The aim of this study was to evaluate the association between smoking status and the presence and progression of AAC in a male population.  Materials and methods: a total of 306 men aged &gt; 50 years who underwent dorsolumbar radiographic imaging, repeated 4 years later, were selected to determine the presence and progression of AAC. Data collected included age, body mass index (BMI), diabetes status, and smoking history (age at initiation, cigarettes per day, and duration of smoking).  Results: current smokers and former smokers showed a higher prevalence of AAC (52.7 % and 52.6 %, respectively) compared with never smokers (25 %), with former smokers being older. After adjustment for age and BMI, current smoking was associated with AAC prevalence (odds ratio [OR] = 3.95; 95 % confidence interval [95 % CI], 2.00-7.82). A similar association was observed in former smokers (OR, 2.93; 95 % CI, 1.56-5.51). Ten years of aging was associated with AAC (OR, 1.64; 95 % CI, 1.35-1.93), representing a 4.6- and 3-fold lower risk compared with being a current or former smoker, respectively. After 4 years of follow-up, both current and former smoking were associated with AAC progression (OR, 2.57; 95 % CI, 1.08-6.14; and OR, 2.34; 95 % CI, 1.07-5.13, respectively). Heavy smoking and longer smoking duration further increased this risk.  Conclusions: smoking status was associated with a higher prevalence and progression of AAC. In conclusion, smoking conferred a greater risk of AAC than aging itself.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[Calcificación aórtica]]></kwd>
<kwd lng="es"><![CDATA[Fumador]]></kwd>
<kwd lng="es"><![CDATA[Exfumador]]></kwd>
<kwd lng="es"><![CDATA[Envejecimiento]]></kwd>
<kwd lng="en"><![CDATA[Aortic calcification]]></kwd>
<kwd lng="en"><![CDATA[Smoker]]></kwd>
<kwd lng="en"><![CDATA[Former smoker]]></kwd>
<kwd lng="en"><![CDATA[Aging]]></kwd>
</kwd-group>
</article-meta>
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